AAS Androgenic to Anabolic ChartHaven't received registration validation E-mail? User Control Panel Log out. Forums Posts Latest Posts. View More Photo Galleries. Forum Themes Elegant Mobile.
AAS Androgenic to Anabolic Chart | cialispanettet.top
Haven't received registration validation E-mail? User Control Panel Log out. Forums Posts Latest Posts. View More Photo Galleries. Forum Themes Elegant Mobile. Medichecks - The stress free, speedy way to get a comprehensive, accurate insight into your health.
Our tests include Sports Hormone checks, testosterone, iron status, cholesterol and more. Essentials Only Full Version. I cant really post the link becuase it might break the rule of giving sources but this comes from GA LABS the board they run has these up and I found it very helpful, I just wanted to share the knowledge.
Guess it also doesn't take into account common doses used either. I wonder how it was measured? I've wondered this myself - I suspect it's a bit of a case of guess work to a large extent. These types of figures are arrived at by using rat models and measuring the growth effect on the levator ani and prostate tissues. I thought Proviron was an androgen?
How come it's anabolic rating is about 3 - 4 times higher than it's androgenic rating? LOL Metribolone, what was the intended purpose of this drug? Only available for research purposes. Originally produced by Negma, but never approved for production. The difference is the attachment of a alpha-methyl group for oral activity. So one could refer to methyltrienolone as oral trenbolone. It was first explored quite some time ago by Negma in France, the same company that marketed Parabolan trenbolone.
But the drug was never approved by the French government and was hence never produced. The reason was extreme hepatoxicity. Bill Roberts, the biochemist, once commented that taking methyltrienolone made taking insane doses of anadrol and Halotestin together look mild on the liver. While I was unable to find anything in the literature that describes the extent of the liver toxicity, it's a generally accepted fact.
That's also why, to the dissapointment of many, you will never find a commercially marketed methyltrienolone product. Its only sold in bulk to labs and universities for research studies involving androgens. Mainly because and those who wish it was available will wish so even more now its such a potent androgen.
There is some conflicting information in that regard however. Organic chemist Patrick Arnold, head of LPJ research, once stated that methyltrienolone was the most powerful steroid ever, and that statement has been blown out of proportion and taken on a life of its own. While androgenically a very potent steroid, methyltrienolone is still basically trenbolone with a alpha-methyl group.
A group that has the tendency to actually reduce the androgenic potency. So it may actually be somewhat milder than trenbolone, on the contrary to what many pseudo steroid guru's are now claiming after reading Pat Arnold's statement.
I can't find any other documented effects of the alpha-alkylation influencing androgen binding in a positive way. It's a potent androgen, with more binding than even DHT2, but the study that claims that is mild at the very best about quantifications, whereas people have used the term times more powerful than testosterone, which is surely exaggerated. What is interesting is that it seems to show nearly no binding for sex-hormone binding proteins, which makes it a popular choice in androgen receptor studies3, since it will demonstrate equal binding in all tissues regardless of the presence and amount of these proteins.
No doubt this plays a role in its supposed binding capacity. In this instance the alpha-alkylation may have played a key role, since it has been demonstrated a multitude of times that alpha-methyl groups decrease the binding for sex-hormone binding proteins as well as most other structures, and due to its triple double bond, trenbolone really didn't bind well to these to begin with.
One of the findings made in clinical tests with methyltrienolone was the discovery of high amounts of the DHT-deactivating enzyme 3alpha-hydroxysteroid dehydrogenase in muscle tissue4. Once again proof that God meant to keep us humans weak. Follow-up studies then went on to show that DHT nonetheless showed similar binding in the prostate, and showing little or no presence of the deactivating enzyme.
So God would rather have us all die of prostate cancer than gain a few ounces of muscle. It's a comforting thought, no? What methyltrienolone, despite its amazing capacity, still doesn't overcome are the basic problems with any 19Nor compound.
First of all its effects on libido. Methyltrienolone still seems to affect our sex drive in such a potent manner that the dreaded Deca Dick temporary impotence is a very real threat5.
Another is that it still binds almost equipotently to the progesterone receptor3. The latter would be of little concern as long as no circulating estrogen is present since methyltrienolone does not aromatize, but could cause problems such as aggravating water retention and gyno growth of breast tissue in men if combined with an aromatizing androgen or an estrogen. While many may wish that an incredibly strong androgenic, non-aromatizing compound as this was available for daily use, its not. And if the indications are true, its probably best.
I've warned many people for the toxicity of fluoxymesterone, and everything points to it that methyltrienolone makes fluoxymesterone look like Tums tablets in terms of liver toxicity. Obviously this section is mostly useless, as any who would use, let alone stack methyltrienolone for any decent period of time, wouldn't really be around long enough to tell us how well it worked.
Ideally one would use it alone, while dieting or for the purpose of gaining lean mass. The androgenic potency is slightly higher than that of trenbolone, so the risk for aggravated hair loss, acne, prostate hypertrophy and deepening of voice is not only realistic, but almost likely. If one were to use it, you would probably have to use every trick in the book to protect your liver and stay alive: The blood pressure raise would not be mild either.
So something to lower blood pressure is advised as well. Methyltrienolone, a specific ligand for cellular androgen receptors. Steroids Aug;26 2: Binding of methyltrienolone to various androgen-dependent and androgen-responsive tissues in four animal species. Horm Res ;7 6: Determination of rat muscles androgen-receptor complexes with methyltrienolone.
Steroids Feb;29 2: Failure of the synthetic androgen 17 beta-hydroxy alpha-methyl-estra-4,9,trieneone methyltrienolone, R to duplicate the activational effect of testosterone on mating in castrated male rats. J Endocrinol Apr; 1: Strongest steroid known to man. Kiss your liver goodbye though. Correct me if Im wrong, but Ive seen these figures before and believe they're calculated from cross-sectional studies of the levitor ani muscle in lab rats?
Whether or not thats gonna translate to real gains in humans is another matter Any such list is badly flawed Unfortunately, there are no studies comparing more than a handful of steroids against each other in terms of androgenicity and effect on muscle growth So a big list like that is just guesswork for the most part.
Create an account or sign in to comment. I've wondered this myself - I suspect it's a bit of a case of guess work to a large extent 5. PartyBoy Moderator Total Posts: The Lakes, UK Status: Newlad Universe Member Total Posts: Genics Pro-Member Total Posts: Bondi Beach, Sydney Status: